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What does an eclipse look like from the moon? Firefly Aerospace’s Blue Ghost lander just sent back a stunning image from the lunar surface.

The commercial space company’s lander, which touched down on the moon without a hitch on March 2 as part of a mission for NASA, took a high-definition image of the eclipse from its top deck in the early hours of Friday — and it’s mighty beautiful.

“Blue Ghost got her first diamond ring! Captured at our landing site in the Moon’s Mare Crisium around 3:30 am CDT, the photo shows the sun about to emerge from totality behind Earth,” Firefly Aerospace wrote online. (A “diamond ring” effect happens during a total solar eclipse when the sun just begins to appear from behind Earth; it also happens just before Earth totally blocks the sun.)

From our perch on Earth, it was a total lunar eclipse, and from the moon it was a total solar eclipse. These unique events happen when the sun, Earth, and moon align, allowing Earth to cast a shadow on the moon and block most sunlight from reaching the lunar surface. But our planet’s atmosphere still allows red wavelengths of light to squeeze through and travel through space, illuminating the moon in reddish, rusty, orangish, or crimson colors.

According to the company, it’s “the first time in history a commercial company will be actively operating on the Moon and able to observe a total solar eclipse where the Earth blocks the sun and casts a shadow on the lunar surface.”

Firefly’s Blue Ghost Mission 1 launched on Jan. 15 and landed on the moon on March 2 after a 45-day trip — and the photos Blue Ghost has been sending back are breathtaking. In the above photo, you can also see the Blue Ghost lander’s NASA equipment, including a Lunar Environment heliospheric X-ray Imager, Lunar Magnetotelluric Sounder mast, and X-band antenna.

.Blue Ghost moon lander just beamed back stunning photo of the eclipse

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An Australian man in his forties has become the first person in the world to leave hospital with an artificial heart made of titanium. The device is used as a stopgap for people with heart failure who are waiting for a donor heart, and previous recipients of this type of artificial heart had remained in US hospitals while it was in place.

The man lived with the device for more than three months until he underwent surgery to receive a donated human heart. The man is recovering well, according to a statement from St Vincent’s Hospital in Sydney, Australia, where the operations were conducted.The Australian is the sixth person globally to receive the device, known as BiVACOR, but the first to live with it for more than a month.

“This is certainly an important development in the field,” says Julian Smith, a cardiac surgeon at the Victorian Heart Institute at Monash University in Melbourne, Australia.

“It is incredibly innovative,” says Sarah Aitken, a vascular surgeon at the University of Sydney, but she adds that there are still many unanswered questions about the level of function that people with it can achieve and the ultimate cost of the device. “This kind of research is really challenging to do because it is very expensive” and the surgery involved is very high-risk, says Aitken.

The latest success will help researchers to understand how people cope with this device in the real world, says Joseph Rogers, a heart-failure cardiologist and president of the Texas Heart Institute in Houston. “They weren’t being constantly monitored by medical teams,” says Rogers, who led the first trial of the device in the United States last year.

In all cases, the BiVACOR was used as a temporary measure before a donor heart became available. Some cardiologists say that it could become a permanent option for people not eligible for transplants because of their age or other health conditions, although the idea still needs to be tested in trials. In the United States, close to 7 million adults live with heart failure, but only about 4,500 heart transplants were performed in 2023, in part because of a shortage of donors.

Suspended rotor

BiVACOR was invented by biomedical engineer Daniel Timms, who founded a company named after the device, with offices in Huntington Beach, California and Southport, Australia.

The device is a total heart replacement and works as a continuous pump in which a magnetically suspended rotor propels blood in regular pulses throughout the body. A cord tunnelled under the skin connects the device to an external, portable controller that runs on batteries by day and can be plugged into the mains at night.

Many mechanical heart devices support the left side of the heart, and typically work by pooling blood in a sack, which flexes some 35 million times a year to pump blood. But these devices have many parts and often suffer failures. BiVACOR, which only has one moving part, will in theory experience fewer problems of mechanical wear, says Rogers.

US trials

The Australian recipient of BiVACOR had severe heart failure, and received the titanium device in a six-hour operation in November. In February, he was discharged from hospital, stayed in a residence close by and led a relatively normal life. In March, he received a donor heart.

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Imagine a world where everyone had brown skin. Tens of thousands of years ago, that was the case, say scientists at Pennsylvania State University. So, how did white people get here? The answer lies in that tricky component of evolution known as a genetic mutation.

Out of Africa

Scientists have long known that Africa is the cradle of human civilization​. There, our ancestors shed most of their body hair around 2 million years ago, and their dark skin protected them from skin cancer and other harmful effects of UV radiation. When humans began leaving Africa 20,000 to 50,000 years ago, a skin-whitening mutation appeared randomly in a sole individual, according to a 2005 Penn State study. That mutation proved advantageous as humans moved into Europe. Why? Because it allowed the migrants increased access to vitamin D, which is crucial to absorbing calcium and keeping bones strong.

“Sun intensity is great enough in equatorial regions that the vitamin can still be made in dark-skinned people despite the ultraviolet shielding effects of melanin,” explains Rick Weiss of The Washington Post, which reported on the findings. But in the north, where sunlight is less intense and more clothing must be worn to combat the cold, melanin’s ultraviolet shielding could have been a liability.

Just a Color

This makes sense, but did scientists identify a bonafide race gene as well? Hardly. As the Post notes, the scientific community maintains that “race is a vaguely defined biological, social, and political concept…and skin color is only part of what race is—and is not.”

Researchers still say that race is more of a social construct than a scientific one because people of purportedly the same race can have as many differences in their DNA as people of separate so-called races do. It’s also difficult for scientists to determine where one race ends and another begins, considering that people of supposedly different races may have overlapping features in terms of hair color and texture, skin color, facial features, and other characteristics.

Members of Australia’s aboriginal population, for example, sometimes have dark skin and blond hair of various textures. They share traits with people of African and European ancestry alike, and they are far from the only group not to fit squarely into any one racial category. Scientists posit that all people are roughly 99.5% genetically identical.

The Penn State researchers’ findings on the skin-whitening gene1 show that skin color accounts for a minuscule biological difference between humans.

“The newly found mutation involves a change of just one letter of DNA code out of the 3.1 billion letters in the human genome—the complete instructions for making a human being,” the Post reports.

Skin Deep

When the research was first published, scientists and sociologists feared that the identification of this skin-whitening mutation would lead people to argue that whites, Blacks, and others are somehow inherently different. Keith Cheng, the scientist who led the team of Penn State researchers, wants the public to know that’s not so. He told the Post, “I think human beings are extremely insecure and look to visual cues of sameness to feel better, and people will do bad things to people who look different.”

His statement captures what racial prejudice is in a nutshell. Truth be told, people may look different, but there’s virtually no difference in our genetic makeup. Skin color really is just skin deep.

Not So Black and White

Scientists at Penn State continue to explore the genetics of skin color. In a 2017 study published in the journal Science, 2 researchers report their findings of even greater variants in skin color genes among native Africans.

The same appears to be true of Europeans, given that, in 2018, researchers used DNA to reconstruct the face of the first British person, an individual known as the “Cheddar man” who lived 10,000 years ago. The scientists who took part in the reconstruction of the ancient man’s face say that he most likely had blue eyes and dark brown skin. While they do not know for sure what he looked like, their findings dispute the idea that Europeans have always had light skin.

Such diversity in skin color genes, says evolutionary geneticist Sarah Tishkoff, the lead author of the 2017 study, likely means that we can’t even speak of an African race, much less a white one. As far as people are concerned, the human race is the only one that matters.

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In 2025, there have been at least three measles outbreaks in the U.S. The biggest outbreak to date is in Texas, with 198 cases, resulting in 23 hospitalizations and one death of a school-aged child, as of March 7, 2025.1 This child had not received the measles vaccine and had no underlying health conditions.

Unfortunately, amid these scary outbreaks, some misinformation about measles treatments and prevention has also been spreading—specifically, the idea that vitamin A can be used to treat and prevent measles.

This has led the the American Academy of Pediatrics (AAP) to recently release online statements warning parents not to rely on vitamin A for measles prevention, and that too much vitamin A can be dangerous for children.

The AAP emphasizes, “measles-mumps-rubella (MMR) vaccination remains the most important tool for preventing measles.” The AAP also underscores how extremely contagious the measles is, noting the virus can remain in the air as many as two hours after someone infected has left the vicinity. As such, their focus is on the importance of vaccination as the primary preventative tool for measles.

Vaccines Are the Best Way To Prevent Measles

Therese Linnon, DO, a pediatrician at Akron Children’s Hospital says while vitamin A can be used to help with some symptoms of the measles once a patient has been diagnosed, it is far better to prevent the infection in the first place by getting the measles vaccine.

“Vitamin A should not be a replacement for the vaccine. There is no dose of vitamin A that will protect them from getting the measles virus,” she explains.

Mahvash Madni, MD, a pediatrician and spokesperson for the AAP agrees, referencing the hundreds of measles-related child deaths in the U.S. every year, prior to the existence of a vaccine.

“Nutrition and a strong immune system are important in helping prevent disease but certain viruses that are very powerful can overwhelm the immune system regardless of our best efforts,” she says. “Measles is one of these viruses. That is why years of research and effort went into coming up with an extremely safe and effective vaccine which was put into effect decades ago.

As per CDC guidelines, the current measles vaccine—the MMR vaccine—should be given in two doses. The first dose should be given when a child is 12-15 months, and the second dose should be given between 4 and 6 years. Two doses of MMR vaccine is 97% effective against measles infections.

Can Vitamin A Be Used To Treat Measles?

First of all, it’s important to understand that the measles isn’t just a virus that causes an annoying rash.

“Children feel and look very ill,” explains Dr. Madni. “It can cause pneumonia, neurological problems like encephalitis and death.”

As Dr. Madni and the CDC note, for every 1,000 children who get the measles, between one and three of them will die.

Moreover, Dr. Madni emphasizes, there are no treatments or “cures” for the measles. “It has to run its course like most viruses,” she says. So why do some people suggest supplementing with vitamin A?

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Parents/rudi_suardi via getty images

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